Sexual Diseases – Scabies, Trichomoniasis and Thrush

A healthy sex brings a lot of joy and gratification whereas unsafe sex with infected people brings a lot of worry and panic in life. If you have had sex with a person, make sure you and your partner are healthy and free from any infection. Safe sex is the best way to protect against sexual diseases.

Scabies, Trichomoniasis and Thrush are common sex related diseases. Here, we will discuss the causes, symptoms and treatment of scabies and trichomoniasis.

1. Scabies is caused by sexual contact with someone who is infected but this is rare. The major symptoms of scabies are itchiness, red raised bumps on the skin, particularly between fingers or under the breasts, around the wrist and on genitals or buttocks.

You can get rid of scabies by treating the whole body with a special lotion which could be purchased from the medical stores. Some people also wash their bed sheets and towels in very hot water to avoid getting scabies again, although the risk of infection in this way is very small.

2. Trichomoniasis is caused by a tiny organism which affects the vagina and urethra. You can get this disease by having sex with someone who is infected. The symptoms of trichomoniasis in women are yellow or white discharge from the vagina which is quite smelly and itchiness in the vaginal area. Men may have no symptoms so they may not know they have got the disease. This disease can be treated by taking special tablets which you can get form your doctor.

3. Thrush can be caused by a yeast infection which you can get by having sex with someone who is infected. But you can also get thrush without sexual contact. It is caused by yeast called Candida albicans which affects the vulva and vagina in women and penis in men.

The major signs and symptoms for a woman that she has got thrush include itching around her vulva and a thick white discharge from her vagina. The vulva may also smell a little and it may hurt to pass urine. The major signs and symptoms for a man that he has got thrush include a sore and itchy penis.

Woman can treat themselves by putting a tampon dipped in plain yogurt into their vagina. If this does not work, they should see their doctor. Doctor may prescribe a cream. Men are usually given a cream.

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Cure For Hepatitis A – Is There a Natural Cure?

Hepatitis A is perhaps the most easily spread Hepatitis virus among the family of fatal Hepatitis viruses. Unlike Hepatitis B and C, which are considered more deadly than the rest of the types of the disease, HAV is spread through infected food and drinking water. Fortunately, there is a natural and herbal cure for hepatitis A.

It is a startling fact that 90 percent of deaths in children less than five years old are caused by diarrhea, a disease that arises due to lack of clean drinking water. As the problem of clean drinking water is massive on the global scale, the threat of spread of Hepatitis A virus, and a cure for hepatitis a is quite obvious. With such high risk of spread of Hepatitis A virus, especially in some parts of the world, it is in your best interest to find out if you are at high risk of getting infected with HAV.

Preventing hepatitis A

Whether you have to frequently catch the plane for international flights for the purpose of business or if you are an enthusiastic tourist who loves to visit world tourist attractions, you have to watch out for those regions where the risk of HAV infection is considered high. Research suggests that if you are a frequent visitor to Mexico, Greenland, Indonesia and neighboring nations and the African continent, you are at high risk to getting infected with Hepatitis A virus. Asia and Eastern European nations have a moderate level risk of HAV spread, while South American continent is another danger zone as far as HAV is concerned.

Again, the reason why the above mentioned regions put you at high risk of getting infected with HAV is that there is poor provision of clean drinking water in those areas. Despite the fact that contaminated water infects millions globally with HAV, as a traveler hitting the road, you should also avoid eating seafood, fruits, vegetables and any other sort of unhygienic food.

A new cure for hepatitis a?

A new cure for this type of hepatitis was recently discovered, and has already been used to treat countless patients, suffering from the hepatitis A infection. It has proven to be extremely effective, although herbal.

Hepatitis Symptoms

Hepatitis is derived from the Greek word “hepat” meaning liver and the Greek suffix “-itis” meaning inflammation. It is characterized by the destruction of liver cells and the presence of inflammatory cells in the liver tissues. It can either be acute or self-limiting where it heals on its own or it can be chronic which is longer or more persistent. There are many causes including:

  • Toxins such as alcohol.
  • Infections.
  • Autoimmune processes which is an immune response against your own cells or tissues.

Often people do not have any symptoms or only develop them further on in the disease so it can be difficult to detect and can be quite advanced before it is actually picked up.

Hepatitis B:

Hepatitis B is inflammation of the liver caused by a DNA virus causing viral hepatitis. At the moment in Western countries only 2% of the population are infected with chronic hepatitis. It is a blood-borne infection which can be transmitted by:

  • Re-use of contaminated needles.
  • Unprotected sexual contact.
  • Blood transfusions
  • During childbirth from the mother to the child if the mother is infected.

There are several vaccinations for Hepatitis B: it can either be made from recombinant DNA technology or obtained from the plasma of patients with long-standing Hepatitis B virus infections. Recombinant DNA technology involves adding the relevant bits of DNA into a bacterium plasmid which is put back into the bacteria which then replicates itself including the new piece of DNA. This can then be used as a vaccine so that people develop their own antibodies against the virus. The vaccine is targeted at people who are most at risk including:

  • Family members of people with Chronic Hepatitis B
  • Sexual partners of people with Hepatitis B
  • Newborn babies of mother with Hepatitis B
  • Drug Users
  • Homosexual men
  • Hospital staff who frequently come into contact with blood.

Symptoms of Acute Hepatitis B:

  • General ill-health
  • Loss of appetite
  • Nausea
  • Vomiting
  • Body aches
  • Mild fever
  • Dark urine
  • Development of jaundice.
    This is a yellowing of the sclera (white parts of the eye) the skin and the mucous membrane. It is caused when bilirubin (a yellow break down product of haem which is an iron containing group) levels in the blood increase. This is an insoluble substance which travels to the liver bound to serum albumin. It is joined with glucuronic acid forming bilirubin diglucuroonide which is more soluble and is excreted from the liver as bile. Unnatural cell death (necrosis) reduces the liver’s ability to make and excrete bilirubin leading to a build up of blood in the liver.

 

Acute Hepatitis B lasts a few weeks and in most people it gradually improves and doesn’t usually require treatment. The incubation period for Hepatitis B is usually 2-6months and 1 in 20 patients develops chronic Hepatitis B while 1 in 5 develops cirrhosis.

Symptoms of Chronic Hepatitis B:

· It can be asymptomatic.

· Chronic inflammation of the liver

· Develop fibrosis which is liver scarring

· Develop cirrhosis which is advanced liver scarring. The symptoms of cirrhosis are:

· Bruising

· Bleeding

· Bone pain

· Enlarged veins (varicose veins) around the abdomen

· Fatty stools

· Jaundice

· Increases the risk of hepatocellular carcinoma or liver cancer.

Chronic Hepatitis B can be diagnosed by using blood or serum tests that detect the presence of the viral antigens which are proteins produced by the virus, or they detect the presence of antibodies which are produced by the host as an immune response. The hepatitis surface antigen is most frequently tested for as it is the first detectable viral antigen.

There are 7 medications that can be used to treat Hepatitis B;

5 antiviral drugs:

  • Lamivudine
  • Adefovir
  • Telbivudine
  • Entecavir
  • Tenofovir

2 immune system modulators:

  • Interferon alpha-2a
  • Pegylated interferon alpha-2a.

95% of infected adults and older children will stage a full recovery and develop protective immunity to the virus.

Hepatitis C:

Hepatitis C is a blood-borne infection caused by the Hepatitis C virus which affects the liver. It is spread by blood to blood contact and at the moment there is no vaccination to protect against it. Acute Hepatitis C is the first six months of the infection. 60-70% of infected people are asymptomatic during this phase but they can suffer:

  • General ill health including:
  • Marked weight loss
  • Fatigue
  • Flu-like symptoms
  • Muscle and joint pain
  • Nausea
  • Depression
  • Sleep disturbances
  • Head aches
  • Loss of appetite
  • Abdominal pain
  • Development of Jaundice.
  • Itching
  • Flu-like symptoms

Since the acute phase is often asymptomatic it is difficult to pick up early and treat so many people have developed more serious problems before they are aware they are infected making it more difficult to treat.

It can be tested for as the virus is detectable in the blood 1-3weeks after being infected and the antibodies which are produced as an immune response can be detected 3-12weeks after being infected but as mentioned above many people do not seek medical help because they don’t have any symptoms. 15-40% of people are able to clear the infection in the acute phase but 60-85% of people develop chronic hepatitis C which is where the infection has lasted more than 6 months. The incubation period is usually 1-6months and 8 in 10 patients develop chronic Hepatitis C while 1 in 3 develops cirrhosis.

Symptoms of Chronic Hepatitis C:

  • Often asymptomatic.
  • Inflammation of the liver
  • Leads to fibrosis which is liver scarring
  • Two thirds progress to cirrhosis (which is advanced liver scarring) within 20-30 years.

The rate of progression is increased by:

  • Increasing age
  • Gender: In males the progression is usually faster
  • Alcohol consumption
  • HIV co-infection
  • Fatty liver

Liver biopsy tests are used as they are best for detecting the amount of scarring and inflammation of the liver. Hepatitis C can also be diagnosed using blood serum tests or serology, which detects the presence of antibodies against the Hepatitis C virus. In 80% of people the antibodies can be detected after 15weeks and in 90% they are detected after 20 weeks. You can also test for the presence of the virus using molecular nucleic acid testing methods which measure the amount of virus present.

In Western countries 90% of the people infected with Hepatitis C were infected through transmission of unscreened blood to blood contact for example:

· Body piercing and tattoos

· Injection drug use

· Nasal inhalation

· Blood products from blood transfusions or organ transplants

· From un-sterilized equipment

· Unprotected sexual contact

· Sharing personal items such as razors, tooth brushes and scissors

Hepatitis C can also be transmitted from an infected mother to her child during childbirth.

At the moment a combination of pegylated interferon alpha and an anti-viral drug called ribavirin are used for 24-48weeks to treat the disease. However, this is physically demanding and in some cases can cause temporary disability.

Smoking and alcohol consumption increases the progress of Hepatitis C virus associated fibrosis, cirrhosis and increases the risk of developing liver cancer. Insulin resistance and metabolic syndrome can worsen the prognosis of hepatitis.

It can be prevented by:

  • Not sharing needles (or other drug paraphernalia)
  • Avoiding unsanitary tattoo methods
  • Avoiding unsanitary body piercing methods
  • Avoiding unsanitary acupuncture methods
  • Not sharing personal items
  • Not having unprotected sex.

How to Remove Tonsil Stones – The Right Doctor Can Help

If you ever have to deal with frequent throat pain associated with tonsil swelling even if you don’t seem to have an infection, you might have what are known as tonsil stones. Since the symptoms they cause never degenerate into anything more serious, medical professionals don’t usually put much emphasis into diagnosing them. However, just because they cannot be as serious threat to your overall health doesn’t mean that the pain and discomfort they cause is fun to live with.

What are tonsil stones exactly?

In your throat around your tonsils are small flaps of flesh that can house the formation of the stones. For some people, these crevices are deeper that for others and are therefore more prone to develop this condition around the lingual and palatine areas of the tonsils. The stones themselves are made up of different types of minerals. In fact, analysing their content can reveal magnesium, calcium, ammonia and carbonate residues, among others.

On average, they are about the size of a pea and weigh about 300 milligrams which is less than the weight of a multivitamin tablet. Since they are usually so small and therefore can’t cause any serious health issues, doctors don’t make a big deal out of them. In some isolated cases they can become much larger in which case it becomes essential to remove them.

Removing them requires the right doctor

While most regular health practitioners don’t make a fuss about them or have a hard time diagnosing them, an ENT doctor (specialized in conditions affecting the ears, nose and throat) does take them more seriously. He will be better at finding them and figuring out what is the best way to get rid of them. If you have symptoms that can indicate the presence of tonsil stones (such as constant pain and discomfort, especially while swallowing) consulting an ENT doctor is the best thing you can do. Depending on the size and location of the stones, your ENT doctor might have to apply a local anaesthetic to make the process fast and painless.

A homemade solution

Some individuals prefer to remove the tonsil stones manually at home. To do this, they use either a pick of cotton swab and proceed to dislodge them from inside the tonsil crevices. This of course requires a mirror and a good light source in order to see exactly where the stones are located and where they are poking around. This way of doing has its downsides though as it can induce quite severe gag reflex making it even harder to get rid of the stones. Not everybody has this reflex though but if you do, it is still better and easier to see the right doctor.

Lookout for bad breath

One final thing to know about tonsil stones is that if you have had them once, they will most likely come back. Since they are caused in part by a certain type of bacteria that is also a source of bad breath, be on the lookout for recurring halitosis. This sign is a strong indicator of tonsil stones and when other symptoms such as pain while swallowing are present, it might be a good time to get them removed.

Baby Vomiting After Eating

When does my doctor need to be involved?

Call your doctor if your baby’s vomiting is persistent or uncontrollable. This includes a baby who continues to vomit despite an empty stomach (called dry heaving) or who continues to vomit over several hours, unable to tolerate any liquids. Call if the colour of the vomit is red, dark brown, or black. These are all colours associated with blood. Green vomit contains bile and is also worrisome – a doctor should evaluate any child who has it.

Anytime you think that the vomiting is becoming projectile, your baby should be evaluated for pyloric stenosis.

What tests need to be done, and what do the results mean?

Vomiting rarely requires medical tests. In the case of bloody or green vomit, however, an X-ray may be helpful to look at the bowels. X-rays can show poor functioning in the intestines.

If there is blood in the vomit, then a complete blood count may be done to make sure that the baby is not losing too much blood. In extreme cases, if there is a significant amount of bleeding associated with the vomiting, then a small camera can be inserted through the mouth into the esophagus, stomach, and intestine in order to look for the source of the blood. This is called endoscopy.

If pyloric stenosis is suspected, then a physical exam may reveal a small round lump where the outlet of the stomach is. This lump, which feels like an olive, represents the actual thick and tight pyloric sphincter. To confirm the diagnosis, your doctor will do an ultrasound. Sometimes babies must drink some milk during the ultrasound so that the liquid can be followed down to the stomach and the outline of the enlarged pyloric sphincter can seen.

What are the treatments?

The main treatment for most types of vomiting is time. If an infection (such as a virus or bacteria) is the cause, then the infection will typically pass and the vomiting will subside. Some infections – such as parasites – require treatment with specific medications.

There are medications available to stop acute vomiting. These medications, called antiemetics, are rarely used in infants but may be recommended if a child is becoming dehydrated. The most common of these is promethazine (also called Phenergan), given in the form of a rectal suppository since the oral form usually will not stay down in a vomiting child.

If pyloric stenosis is the cause of the vomiting, then the treatment is surgery. The surgery is a relatively simple procedure whereby the thickened, tight pylorus is cut to release the pressure on the outlet of the stomach. Children are often drinking within hours of the surgery, and the forceful spitting up resolves completely.

What are the possible complications?

The most worrisome complication of persistent vomiting or longstanding pyloric stenosis is dehydration. Dehydration can become quite severe, especially in a small baby with little reserve.

Repetitive or forceful vomiting can also cause tears in the lining of the esophagus. When this occurs at the lower end of the esophagus, it is called a Mallory-Weiss tear. Its hallmark is bright-red blood with vomiting.

Food Poisoning From Botulism

While food poisoning can be caused by a variety of viruses, bacteria, and parasites that can come to inhabit food products via unsafe handling or preparation techniques, we often do not know the exact perpetrator of your illness. However, one dangerous source of food poisoning is the Clostridium botulinum bacteria.

When you eat food inhabited by Clostridium botulinum or its byproducts, you can contract the illness called botulism. The symptoms of botulism include double vision, drooping eyelids, slurred speech, difficulty swallowing, and muscle weakness. Thus, botulism is typically an easily recognizable type of food poisoning.

The reason why this type of food-borne diseases causes muscular weaknesses rather than the typical nausea, vomiting, diarrhea, and abdominal pain is that Clostridium botulinum produces a neurotoxin that interferes with your muscle function. In fact, botulinum neurotoxin is considered an extremely dangerous and potent substance. Scientists have estimated that one gram of the toxin could kill up to one million people, which is why some people have talked about the dangers of terrorists potentially using botulinum toxin for bioterrorism. Interestingly, though, many people choose to purposely inject themselves with this toxin in order to lessen the appearance of wrinkles. That’s right-botulinum toxin is the basis for the facial injection Botox.

The toxin from Clostridium botulinum actually paralyzes the nerves so that they lose the ability to signal the muscles into contracting. Because the damage done to the nerve can be semi-permanent, the effects of botulinum toxin can be very long-lasting.

When you consume food tainted with botulism, you should visit your doctor or emergency room as soon as you begin to lose muscular control and think that you have botulism. Doctors can inject and antitoxin that will help prevent the spread of the neurotoxin and stop the damage being done to your nerves. While the antitoxin can stop the toxin from spreading, it may take you weeks to recover, as mentioned above.

One serious complication resulting from botulism is respiratory distress. Because it takes so long to recover from this condition, people who suffer from respiratory damage due to the botulinum toxin may have to subsist on breathing machines for several months. Indeed, it may take years for you to get over your fatigue and shortness of breath.

Botulism can be prevented with proper canning and food handling techniques. If you ever suspect that a canned good has botulism, it is always better to throw it away than risk contracting botulism. Also, be sure to cook all of your food thoroughly to kill Clostridium botulinum and destroy its toxin.

If you or someone you know has suffered from the terrible effects of botulinum toxin, you should contact a product liability lawyer about your rights. For more information, check out the product liability attorneys from Friedman & Bonebrake, PC today.

Toothpaste and Mouth Ulcers – Is There a Link?

Mouth ulcers and other common mouth conditions

Nearly everyone will remember suffering from a mouth ulcer at some point in their lives, whilst around 20% of people experience recurrent mouth ulcers. These have often been linked with anxiety, eating sharp foods which damage the tissue of the mouth and women’s hormonal changes. Certain foods have also been highlighted as triggering mouth ulcers; these include strawberries, cheese and coffee.

 

Bleeding gums are also a very common condition, again one which most people will experience. It often occurs during brushing where the gums are not as healthy as they could be, and when it occurs alongside inflammation, is generally understood as a sign of gingivitis.

Gingivitis is more commonly known as gum disease. It is generally caused when plaque develops on the teeth, giving bacteria a chance to proliferate and release toxins into your mouth. These toxins then irritate your gums, making them sore and inflamed, and giving you bad breath. Left untreated, it can develop into a more serious condition known as periodontitis.

The generally recommended way to solve these conditions is to brush your teeth thoroughly twice a day with a fluoride toothpaste, as well as to floss to keep the areas between your teeth as clean as possible.

 

Could SLS in toothpaste be to blame?

Whilst this solution does work for many people, some people suffer from recurrent mouth problems despite keeping a good oral health routine. Could it be that there is a link between their toothpaste and mouth ulcers, bleeding gums or gum disease?

 

There is an ingredient known as Sodium Lauryl Sulphate (SLS) which is added to many of the toothpastes you will find on your local supermarket shelves. Next time you go shopping, have a look and see just how many of the “leading brands” use it. SLS is used in toothpastes to make them foam when you brush your teeth. 

 

SLS is also used in a multitude of other toiletries, including shower gels, shampoos and even hand washes, as it is such an effective and cheap foaming agent. Unfortunately, it is also a skin irritant. It is widely recognised as being such and is even used in clinical studies as a standard skin irritant against which other potential irritants are compared.

 

The mouth is a sensitive area of the body and it is only natural that what can irritate our skins can also irritate our mouths. SLS has been shown to cause damage to oral tissues and reaction to SLS may cause gingivitis, receding gums and canker sores, a type of mouth ulcer.

 

Given this information, putting SLS in toothpaste doesn’t seem like such a good idea. It also makes a lot of sense that this might be the problem for those who have recurrent oral issues but who maintain a good teeth cleaning routine. 

 

SLS-free toothpaste gave 60% mouth ulcer reduction

One study, although admittedly very small, found a 60% reduction in ulcers when people with recurrent mouth ulcers switched to using an SLS-free toothpaste. Carried out by a research team from the University of Oslo, this study demonstrates that SLS in toothpaste and mouth ulcers could well be linked.

 

Whilst this is just one small piece of research, it is indicative of the growing weight behind the idea that toothpaste might be one cause of sore gums, mouth ulcers and gingivitis. SLS is by no means the only cause of these conditions, as oral hygiene certainly has its part to play, but for some it may be the cause of long-standing problems.

 

More and more people who suffer from these uncomfortable oral conditions are now looking to buy SLS-free toothpaste to see if an SLS reaction is behind their mouth problems.

 

It is not known how many people have problems with SLS in toothpaste and have sore gums and ulcers as a result. However, buying an SLS-free toothpaste is a very cheap and easy first port of call (after visiting your dentist, of course) if you are looking to find a solution to mouth ulcers or sore gum problems. 

 

SLS in toothpaste and other toiletries

As mentioned earlier, the SLS in toothpaste is also found in many other toiletries. Its use can lead to skin sensitivity and discomfort in many people, yet it is considered an acceptable ingredient for mainstream skin care and hair care products.

 

Unfortunately, there are also other chemicals in toiletries which, like SLS, may have negative effects on consumers and thus are worth avoiding. These include parabens, phthalates, DEA, TEA and ethanol. An Australian scientist in 2009 identified ethanol in mouthwash as being a ‘significant risk factor’ for oral cancers, as it helps cancer-causing substances to pass through the lining of the mouth.

Indigestion, Acid Reflux, Heartburn – Do You Need To See Your Doctor?

Probably three out of every four people who suffer from indigestion never seek medical advice: they relieve their symptoms by a few changes to their lifestyle and every now and then buy over-the-counter treatments, such as antacids or acid blocking drugs, from the chemist.

When to take action

The aim of this article is to help you to decide whether and when to consult your doctor. You should make an appointment if any of the three following descriptions applies to you.

Sinister symptoms

See Your GP without delay if you have any symptoms of the kind doctors call ‘sinister’, by which they mean symptoms that might be caused by a serious disease such as stomach cancer.

Early diagnosis and treatment give the best chance of a cure, so get prompt medical advice if you experience any of the following symptoms:

  • Unexplained weight loss.
  • Loss of appetite.
  • Difficulty swallowing.
  • Vomiting blood or a brown material that bears resemblance to ground coffee.
  • Passing altered blood in the motions.
  • Indigestion while you are taking non steroidal ant inflammatory drugs.

No improvement

Although indigestion without these sinister symptoms can sensibly be treated at home in the first instance by changes to your lifestyle and over-the-counter remedies such as antacids, you should not persist if there is no improvement. Consult your doctor if your symptoms have not cleared up within two weeks of starting self treatment.

Unusual symptoms

You should consult your doctor if you develop indigestion for the first time in your life after the age of 40, or if you develop a type of indigestion that you have not had before. Your doctor may need to arrange various tests and investigations before beginning treatment

Herbal Remedies For Acid Indigestion

Herbal remedies for acid indigestion can be considered if you do not want to take antacids. Generally, as herbal remedies are natural, they are less harmful to our body. It is true that indigestion is becoming a pervasive problem in modern society. You just need to look around to get an indication. If you look at supermarket and drugstore shelves, you can find them filled with antacids. Television or radio commercials have also been heavily promoting antacids as the cure for heartburn.

An acid indigestion that persists or worsens, despite various solutions you try or adjustments you make in your diet, should not be left unattended. You should have it checked by a health professional.

To achieve proper digestion, your stomach needs to be relaxed. If you tend to overeat or eat too quickly, you are likely to suffer from indigestion. Consuming too much food at one sitting burdens the stomach; its digestive juices get diluted making them function less efficiently. Thus, you may taste the stomach acid and feel pain.

You can try herbal remedies for acid indigestion as safer alternatives to antacids. Herbal remedies have virtually little or no toxicity unlike manufactured over-the-counter medicines. These does not mean that herbs are not potent at all; if fact, some of them are. Before using any natural remedies, you should check with your doctor that it is safe for you to do so. Here are some known herbal remedies for acid indigestion:

Slippery Elm. This herb was used by early folk healers as a digestive tonic and for treatment of acid indigestion and dysentery. The relief it brings to acid indigestion derives from its healing action on the mucous membranes. Herbalists believe it soothes inflamed tissues and draws toxins and other irritants from body tissues.

Licorice. A form of licorice called deglycyrrhizinated licorice (DGL), a chewable form of the herb, is a natural antacid. Unlike a regular licorice, DGL does not have hormonal side effects. This makes DGL more effective and will not contribute to elevated blood pressures, which can be a side effect of other forms. Licorice helps fight ulcerations caused by hyperacidity.

Mint. An ancient medicinal herb, mint is a cooling diaphoretic that relieves indigestion, gas or colic, and heartburn. It can also calm nausea and vomiting. The menthol in mint appears to soothe the smooth muscle lining of the digestive tract. Some studies show peppermint also may help to prevent stomach ulcers and stimulate bile secretions.

Gentian. Gentian contains a chemical (gentianine) that stimulates the secretion of stomach acid, lending some credence to its 3,000-year old history as a digestive aid. Try it before meals. Gentian tastes very bitter, so you might want to add honey to your decoction.

Papaya (Fruit and Leaves). While papaya is not known as a herb, its plant has a surprising potency in combating digestive disorders and calming down a disturbed gastrointestinal tract. Its powerful enzyme, papain, helps to breakdown complex proteins, reducing the digestive load on the stomach.

In addition to the above suggestions, you can also try steamed cabbage. The glutamine in cabbage apparently settles an irritated stomach. Fresh cabbage juice, drank immediately after preparation, also helps in soothing. You should always chew your food well and eat in a stress-free setting to prevent acid indigestion.

Bile Reflux or Acid Reflux?

Although carbonated beverages cause acid reflux, this isn’t the only problem that some acid reflux sufferers are faced with. Bile reflux is another uncomfortable backflow of fluid that often accompanies acid reflux. However, instead of thrusting stomach acid back into the esophagus as is the case with acid reflux, bile reflux throws bile (a digested fluid that is made by the liver) up from the small intestine into the stomach and esophagus, causing inflammation to both.

Due to the fact that bile reflux and acid reflux can occur together, this means that the esophagus is doubly assaulted, which causes more inflammation to its lining, and puts a person at a higher risk for developing complications.

What are the symptoms of bile reflux?

– The signs and symptoms associated with bile reflux are similar to acid reflux, making it difficult to distinguish one from the other, especially when both conditions tend to occur simultaneously. That being said, unlike acid reflux, bile reflux causes inflammation within the stomach, which creates a biting, or burning pain in the upper part of the abdomen.

Other symptoms that are characterized by the condition can include:

– Frequent heartburn

– Nausea

– Vomiting bile

– An occasional cough or croakiness in the throat

Along with symptoms, bile reflux teamed with acid reflux can eventually create complications including:

– Gastritis – This is a complication that is caused by bile reflux alone. Gastritis is characterized by irritation and inflammation within the stomach. Although this isn’t typically a serious condition, in some cases it can cause stomach ulcers, bleeding, and chronic gastritis increases the risk of stomach cancer.

– GERD (gastroesophageal reflux disease) – Frequent attacks of heartburn may be a sign of GERD. This is when a person suffers from chronic acid reflux which can be a potentially serious issue as it may lead to a condition known as esophagitis – the inflammation of esophageal tissue.

– Barrett’s esophagus – This is a condition that occurs after long term exposure to stomach acid and/or bile and results in a change of color and tissue composition in the lower esophagus. The new cells are resistant to stomach acid but they have an increased risk of becoming cancerous.

– Esophageal stricture – Scar tissue can form in the lower esophagus, which results from frequent exposure to stomach acid and/or bile. The scar tissue can cause a stricture (a narrowing in the tube) which can lead to trouble swallowing and increase the risk of choking.

– Esophageal cancer – When the esophagus has been exposed to prolonged repetitive stomach acid and/or bile, cancer has the potential to form practically anywhere along the length of the esophagus. This is a serious and difficult form of cancer to treat.

How do you treat bile reflux and acid reflux together?

Proton Pump inhibitors – The best way to treat these conditions, especially for those who suffer from GERD and Barrett’s esophagus, is proton pump inhibitors. These are medications that are designed to block acid production. These meds can sometimes also help reduce the effects of bile reflux.

Ursodexycholic acid – This is the most common medication for treating bile reflux. Ursodexycholic acid helps to encourage bile flow.

Other medications – If bile reflux is the result of the stomach taking too long to empty, other drugs may be prescribed to improve the flow of food through the stomach

The real trouble with bile reflux is that it is hard to control. Unlike acid reflux which can be managed through diet and lifestyle changes, bile reflux can really only be controlled through specific medications or by surgery in severe cases. Unfortunately, sometimes even after treatment, bile reflux continues to plague sufferers. Thus, bile reflux may need to be treated separately from acid reflux.

Chronic Renal Failure

Individuals with chronic renal failure and uremia show a constellation of symptoms, signs, and laboratory abnormalities additionally to those observed in acute kidney injury. This reflects the long-standing and progressive nature of their renal impairment and its results on many kinds of tissues.

Thus, osteodystrophy, neuropathy, bilateral little kidneys shown by abdominal ultrasonography, and anemia are typical initial findings that recommend a chronic course for a individual newly diagnosed with renal failing about the basis of elevated BUN and serum creatinine.

One of the most typical cause of continual renal failing is diabetes mellitus, adopted closely by hypertension and glomerulonephritis. Polycystic kidney disease, obstruction, and virus are among the less typical brings about of chronic renal failing. The pathogenesis of acute renal disease is very different from that of continual renal illness.

Whereas acute injury towards the kidney results in death and sloughing of tubular epithelial cells, frequently followed by their regeneration with reestablishment of regular architecture, continual injury results in irreversible loss of nephrons. Being a outcome, a greater practical burden is borne by fewer nephrons, manifested as an improve in glomerular filtration pressure and hyperfiltration.

For factors not nicely understood, this compensatory hyperfiltration, which can be thought of being a form of “hypertension” at the level of the individual nephron, predisposes to fibrosis and scarring (glomerular sclerosis). Being a outcome, the rate of nephron destruction and reduction raises, therefore speeding the progression to uremia, the complicated of symptoms and signs that occurs when residual renal purpose is inadequate.

Owing towards the tremendous practical reserve of the kidneys, up to 50% of nephrons could be lost without any short-term evidence of functional impairment. This is why people with two healthy kidneys are able to donate a single for transplantation. When GFR is further reduced, leaving only about 20% of initial renal capability, some degree of azotemia (elevation of blood vessels levels of products usually excreted by the kidneys) is noticed.

Nevertheless, patients might be largely asymptomatic simply because a new constant state is achieved in which blood vessels levels of those products are not higher sufficient to cause overt toxicity. However, even at this apparently stable level of renal purpose, hyperfiltration-accelerated evolution to end-stage chronic renal failure is in progress.

Furthermore, simply because individuals with this level of GFR have small practical reserve, they can very easily become uremic with any additional tension (eg, virus, obstruction, dehydration, or nephrotoxic medicines) or with any catabolic state connected with increased turnover of nitrogen-containing products with reduction in GFR.

The pathogenesis of continual renal failure derives in part from the mixture from the poisonous results of (1) retained products usually excreted by the kidneys (eg, nitrogen-containing items of protein metabolic process), (2) regular products for example hormones now present in elevated amounts, and (3) lack of normal products of the kidney (eg, loss of erythropoietin).

Excretory failure outcomes also in fluid shifts, with increased intracellular Na+ and drinking water and decreased intracellular K+. These alterations may contribute to subtle alterations in purpose of a host of enzymes, transport systems, and so on. Patients with chronic renal failing typically have some degree of Na+ and water excessive, reflecting loss of the renal route of salt and water excretion.

A moderate degree of Na+ and drinking water excess might happen without having objective indicators of extracellular fluid excessive. However, continued excessive Na+ ingestion contributes to congestive heart failure, hypertension, ascites, peripheral edema, and weight gain. About the other hand, excessive drinking water ingestion contributes to hyponatremia.

A typical recommendation for the patient with continual renal failing is to prevent excessive salt intake and to restrict fluid intake to ensure that it equals urine output plus 500 mL (insensible losses). Further adjustments in amount standing can be made either through using diuretics (in a patient who nevertheless makes urine) or at dialysis.

Because these individuals also have impaired renal salt and water conservation mechanisms, they’re a lot more sensitive than normal to sudden extrarenal Na+ and water losses (eg, vomiting, diarrhea, and increased sweating with fever). Under these circumstances, they a lot more easily create ECF depletion, additional deterioration of renal purpose (which may not be reversible), and even vascular collapse and shock.

The symptoms and indicators of dry mucous membranes, dizziness, syncope, tachycardia, and decreased jugular venous filling suggest progression of amount depletion. Hyperkalemia is a severe problem in chronic renal failing, particularly for individuals whose GFR has fallen under 5 mL/min. Above that level, as GFR falls, aldosterone-mediated K+ transportation in the distal tubule increases inside a compensatory fashion.

Thus, a patient whose GFR is between 50 mL/min and 5 mL/min is dependent on tubular transport to maintain K+ balance. Treatment with K+-sparing diuretics, ACE inhibitors, or -blockers-drugs that may impair aldosterone-mediated K+ transport-can, therefore, precipitate dangerous hyperkalemia in a individual with chronic renal failure.

Individuals with diabetes mellitus (the primary trigger of continual renal failure) may have a syndrome of hyporeninemic hypoaldosteronism. This syndrome is really a situation in which lack of renin manufacturing by the kidney diminishes the levels of angiotensin II and, therefore, impairs aldosterone secretion.

As a outcome, impacted individuals are unable to compensate for falling GFR by enhancing their aldosterone-mediated K+ transportation and, therefore, have relative difficulty handling K+. This difficulty is usually manifested as hyperkalemia even before GFR has fallen under 5 mL/min.

Finally, not only are patients with chronic renal failure a lot more susceptible towards the effects of Na+ or amount overload, but they are also at greater risk of hyperkalemia in the face of sudden loads of K+ from either endogenous sources (eg, hemolysis, virus, trauma) or exogenous sources (eg, stored blood vessels, K+-rich foods, or K+-containing medications).

The diminished capacity to excrete acid and generate base in continual renal failing results in metabolic acidosis. In most instances when the GFR is above 20 mL/min, only reasonable acidosis develops prior to reestablishment of a new constant state of buffer production and usage. The fall in blood vessels pH in these people can usually be corrected with 20-30 mmol (2-3 g) of sodium bicarbonate by mouth every day.

Nevertheless, these individuals are extremely susceptible to acidosis within the event of a sudden acid load or the onset of problems that improve the generated acid load. Several problems of phosphate, Ca2+, and bone metabolic process are noticed in continual renal failing as a result of a complex series of events.

The key factors in the pathogenesis of those problems include (1) diminished absorption of Ca2+ from the gut, (a couple of) overproduction of PTH, (three) disordered vitamin D metabolism, and (4) chronic metabolic acidosis. All of these factors contribute to enhanced bone resorption.

Hypophosphatemia and hypermagnesemia can happen via overuse of phosphate binders and magnesium-containing antacids, even though hyperphosphatemia is more typical. Hyperphosphatemia contributes towards the improvement of hypocalcemia and thus serves as an additional trigger for secondary hyperparathyroidism, elevating blood PTH levels.

The elevated blood vessels PTH additional depletes bone Ca2+ and contributes to osteomalacia of chronic renal failing (see later discussion). Congestive heart failure and pulmonary edema can develop in the context of amount and salt overload.

Hypertension is a typical finding in chronic renal failing, also generally on the basis of fluid and Na+ overload. However, hyperreninemia is also a recognized syndrome in which falling renal perfusion triggers the failing kidney to overproduce renin and thereby elevate systemic blood stress.

Pericarditis resulting from irritation and inflammation from the pericardium by uremic toxins is a complication whose incidence in continual renal failure is decreasing owing to earlier institution of renal dialysis. Increased cardiovascular risk is a complication seen in patients with chronic renal failure and remains the leading trigger of mortality in this population.

It results in myocardial infarction, stroke, and peripheral vascular disease. Cardiovascular risk factors in these patients include hypertension, hyperlipidemia, glucose intolerance, chronic increased cardiac output, and valvular and myocardial calcification being a consequence of increased Ca2+ x PO43 product as nicely as other, less well-characterized factors from the uremic milieu.

Individuals with continual renal failing have marked abnormalities in red blood cell count, white blood vessels cell purpose, and clotting parameters. Normochromic, normocytic anemia, with signs and symptoms of listlessness and simple fatigability and hematocrit levels typically within the range of 20-25%, is a consistent function.

The anemia is due chiefly to lack of production of erythropoietin and lack of its stimulatory effect on erythropoiesis. Thus, individuals with chronic renal failure, regardless of dialysis standing, show a dramatic improvement in hematocrit when treated with erythropoietin (epoetin alpha).

Additional causes of anemia may include bone marrow suppressive effects of uremic poisons, bone marrow fibrosis due to elevated blood vessels PTH, toxic effects of aluminum (from phosphate-binding antacids and dialysis solutions), and hemolysis and blood loss associated to dialysis (while the individual is anticoagulated with heparin).

Individuals with chronic renal failure show abnormal hemostasis manifested as elevated bruising, increased blood vessels reduction at surgery, and an elevated incidence of spontaneous GI and cerebrovascular hemorrhage (including both hemorrhagic strokes and subdural hematomas).

Laboratory abnormalities include prolonged bleeding time, decreased platelet element III, abnormal platelet aggregation and adhesiveness, and impaired prothrombin usage, none of that are totally reversible even in well-dialyzed individuals. Uremia is connected with elevated susceptibility to infections, considered to be because of to leukocyte suppression by uremic toxins.

The suppression appears to become higher for lymphoid cells than neutrophils and seems also to affect chemotaxis, the acute inflammatory response, and delayed hypersensitivity more than other leukocyte functions. Acidosis, hyperglycemia, malnutrition, and hyperosmolality also are considered to contribute to immunosuppression in continual renal failing.

The invasiveness of dialysis and the use of immunosuppressive medicines in renal transplant individuals also contribute to an increased incidence of infections. CNS signs and symptoms and indicators might variety from mild sleep disorders and impairment of mental concentration, lack of memory, errors in judgment, and neuromuscular irritability (manifested as hiccups, cramps, fasciculations, and twitching) to asterixis, myoclonus, stupor, seizures, and coma in end-stage uremia.

Asterixis is manifested as involuntary flapping motions seen when the arms are extended and wrists held back to “stop visitors.” It’s because of to altered nerve conduction in metabolic encephalopathy from the broad range of brings about, including renal failure.

Peripheral neuropathy (sensory higher than motor, lower extremities higher than upper), typified through the restless legs syndrome (poorly localized sense of discomfort and involuntary movements from the lower extremities), is a common discovering in continual renal failing and an important indication for starting dialysis.

Patients receiving hemodialysis can develop aluminum toxicity, characterized by speech dyspraxia (inability to repeat words), myoclonus, dementia, and seizures. Likewise, aggressive acute dialysis can outcome in a disequilibrium syndrome characterized by nausea, vomiting, drowsiness, headache, and seizures inside a individual with really high BUN amounts.

Presumably, this really is an impact of rapid pH or osmolality alter in ECF, resulting in cerebral edema. Nonspecific GI findings in uremic patients include anorexia, hiccups, nausea, vomiting, and diverticulosis. Even though their precise pathogenesis is unclear, many of these findings improve with dialysis. Ladies with uremia have reduced estrogen amounts, which perhaps explains the high incidence of amenorrhea and also the observation that they hardly ever are capable to carry a pregnancy to term.

Regular menses-but not a higher rate of productive pregnancies-typically return with frequent dialysis. Similarly, low testosterone levels, impotence, oligospermia, and germinal cell dysplasia are common findings in males with continual renal failing. Lastly, continual renal failure eliminates the kidney as a website of insulin degradation, thereby increasing the half-life of insulin.

This typically has a stabilizing effect on diabetic patients whose blood glucose was previously hard to control. Skin modifications arise from numerous from the results of continual renal failure currently discussed.

Patients with continual renal failing may show pallor because of anemia, skin color changes related to accumulated pigmented metabolites or even a gray discoloration resulting from transfusion-mediated hemochromatosis, ecchymoses and hematomas being a result of clotting abnormalities, and pruritus and excoriations being a outcome of Ca2+ deposits from secondary hyperparathyroidism. Lastly, when urea concentrations are extremely higher, evaporation of sweat leaves a residue of urea termed “uremic frost.”

Lymphedema of Legs – Causes and Treatments?

For the most part, lymphedema of legs or leg lymphedema, as it is most often called, was thought to be due to heredity. Nonetheless, there are other causes. With surgeries for different types of cancers resulting in more cures, there is an increase in lymphedema.

Prostate, ovarian, lung, abdominal and liver cancer surgeries can result in swelling if the removal of lymph nodes in the lower body is performed. Other treatments can cause protein levels to decrease and this, in turn results in fluid leaking into the legs. Swelling may occur only in the ankles and feet or it can be in the calf or thighs.

Lymphedema pumps, which are considered a breakthrough in treating this disorder, have been quite beneficial. Lymphedema of legs can be treated by placing the sleeve over the leg. The sleeve, which is similar to a bandage, inflates and deflates and forces the accumulated fluid to return to the lymph system. This is comparable to the normal function of the lymphatic system.

This treatment can be used once or twice daily for relief from swelling, pain and discomfort associated with lymphedema. The great thing about using a lymphedema pump is that it can be used in the comfort of your home. This eliminates the need to travel to a specialist two or three times a week or more. The lymphedema pump is even portable, so it may be taken with you – wherever you need to go.

Lymphedema of legs can be even more dangerous if left untreated. The build-up of fluid can cause sores to form that will not heal. The possibility of gangrene is present. This could result in amputation of the leg. It is imperative that the lymphatic system removes this fluid and flushes it back into the lymphatic system.

Millions of people suffer from this disorder. Although research has been and continues to be ongoing, there is not a cure for lymphedema yet. The development of the lymphedema pump is an exciting breakthrough for people suffering from this ailment. It has been crucial in managing this condition. Patients have experienced relief from the pain and are able to do things they could not.

Simply walking from one room to another is difficult for those with lymphedema of legs. They often are not able to dress themselves, go to the grocery store or perform the tasks that most people take for granted. The pain is often so intense that they need pain medication. Affecting the normal, everyday tasks that are part of their lifestyle, this is a condition that is not only painful and uncomfortable, but it can be very stressful.

Lymphedema of legs generally affect both men and women. Men who have had prostate surgery or women who have had surgery for cervical cancer are more at risk. There is not a time limit for developing this problem. It can occur immediately after surgery or 20 years later.

Being aware of the symptoms and staying apprised of treatments that can be effective at controlling lymphedema of legs is the most important thing. The earlier it is detected, the more successful the management of the symptoms.

Golfing After a Heart Attack Or Heart Surgery

Commencing cardiac rehabilitation program is an important first step in your goal to getting back to playing golf. During your cardiac rehabilitation program you will learn about controlling your modifiable risk factors to heart disease. This will also include starting an exercise program. You will undergo a fitness test at which they will find out your current level of fitness. A cardiac rehabilitation program will calculate your level of fitness or MET level and show you how to increase your level of fitness safely and effectively.

When we look at any physical activity we gage this measure in METs which stand for metabolic equivalents. METs are a unit in which we use to calculate the level of physical activity or energy expended. One MET is the energy it takes to sit quietly. Therefore, if walking takes 3.5 METs this means that you are going 3.5 times your resting level. Golfing can take anywhere from 3.5 METs to 4.5 METs, therefore it is important to increase your level of fitness with your exercise program.

If you have had recent heart surgery, you should be visiting your surgeon within 3 months to ensure that all the wounds are undergoing the normal healing process. As they start to heal you can start to do some stretching for golf. Please consult your cardiac rehabilitation specialist to ensure that you are doing things safely and slowly. This includes flexibility, aerobic exercises, and resistance training. All three are important in getting you back to your love of golf.

Aerobic exercise is the best form of exercises to increase your fitness level and improve your heart condition. Aerobic means “with oxygen” so you are using oxygen as your primary fuel source to allow the exercise to continue. This includes, dependent on your fitness level, walking, brisk walking, jogging, biking, swimming, cross country skiing, etc. It is important to focus on aerobic exercise as it gives you a base of support for the activities you wish to do such as golfing.

Once you have developed a strong base of support with aerobic exercises then you can proceed to doing strength/resistance training exercises to increase your strength to handle the swing speed of your club during a golf swing.

Performing cardiac yoga can also increase your strength, flexibility and develop a positive mental mindset to get you back to swinging your clubs.

To get back to golfing safely: 1. Enroll in a cardiac rehabilitation program. 2. Start an aerobic exercise program. 3. Get started with some stretching. 4. Re-visit with your cardiologist and/or cardiothoracic surgeon. 5. Start a strength training program. 6. Try cardiac yoga. 7. Start out slow. Try the driving range or nine holes and before you know it you will get back to your regular game or even better.

Once you are ready to golf, start out slow. I always recommend before even picking up a club and swing warm up well. This includes an aerobic warm up, whether it be biking or walking for 5 minutes. The next step would be performing a series of warm up exercises. This includes stretching along with some dynamic warm up exercises. Please consult a qualified cardiac rehabilitation specialist for a list of exercises to get you back to golf.

Golfing is a great and popular activity. If you have heart disease you can get back to playing golf, it just becomes important to know your limits and continue to modify your modifiable risk factors (please read my article or blog about risk factors). Watch out FORE heart disease.

Dental Care and Heart Disease – How it Can Affect You

Dental care and heart disease aren’t two things that you would think would be associated together. It seems as though the distance between these two topics is about a mile wide, however, the facts are in and we now must be on alert in order to avoid what could be devastating consequences.

Your dental hygiene can have an affect on your cardiovascular health if you have open sores or bleeding in your mouth that is there regularly and left untreated. This directly affects those individuals that have bleeding of the gums, better known as gingivitis.

Your mouth naturally produces a substance known as tarter. It is a white chalky substance that develops in our mouths naturally. This tarter can harden and turn into plaque if you don’t use good oral hygiene by brushing and flossing regularly. However, there’s simply no way to avoid some of this substance getting into your blood stream if you have an open wound in your mouth.

The same plaque that develops on your teeth over time can do the exact same thing in your arteries. Over time this can cause blockage of blood flow and eventually may result in a heart attack or stroke.

So what can you do to decrease the odds of this happening to you?

The first thing you must do is schedule an appointment with your dentist. Next, your dentist will likely sit you down and have this same discussion with you. He or she will explain the risks of not getting treated and exactly what treatment involves.

You’ll need to get an in-depth oral examination, as well as x-rays so that the dentist can check the condition of your teeth and gums. Your dentist will likely want to do a thorough cleaning of your teeth to remove any plaque that has built up over time. This will also greatly reduce any bleeding of your gums and help them to heal.

You’ll receive a follow up appointment so any other fillings, etc can be addressed.

Once you have your teeth and gums back in a state of good repair then you’ll want to maintain good personal hygiene. You can do this by brushing your teeth twice a day, flossing daily, avoiding sweets and following up with your dentist every 6 months for check-ups and cleaning.

Common Cichlid Diseases To Be Aware Of

As a fish owner, you must realize that they do not remain healthy all the time and tend to get sick. It is challenging caring for the sick fish. However as a cichlid grower, it is your responsibility to tend to them and ensure that they regain back their good health. If you neglect the initial symptoms of cichlid diseases, you may cause symptoms to worsen and this may also lead to death.

Cichlids get sick for various reasons including parasite, fungal and bacterial infections apart from poor quality of water and other environmental factors.

Watch out for these common cichlid diseases –

Malawi bloat – Fishes on a vegetable diet and African cichlids are commonly affected by the Malawi bloat disease. Symptoms of this disease include white faeces, stomach swelling, loss of appetite, sulking at the tank bottom and rapid breathing. If this cichlid disease progresses, in its advanced stage could cause swim bladder, kidney and liver damage and lead to the death of the cichlid within 72 hours.

According to some, a protozoan found in the fish intestines especially when they are kept in dirty water or if they are not fed properly, is one of the major causes of Malawi bloat. If you find your cichlid suffering from this disease, you must remove filters, change at least fifty percent of the water and add Clout and Metronidazole to the water.

Swim bladder disease – The epithelium lining of the abdominal sac that is mainly responsible for buoyancy of the fish is affected in this cichlid disease. Affected cichlids find it impossible to stay at the tank bottom and tend to float on the top.

Major causes for this disease include poor diet that leads to blockage, bowel irritation, intestinal gas and parasite and bacterial havoc on swim bladder. This disease can be controlled by avoiding feeding too much dried food or protein and introducing fibre rich foods like lettuce, carrots, spinach, peas, squash and zucchini.

Cotton wool disease – Symptoms of this cichlid disease include damaged fins, ulcer, whitish gray skin coating, head tissue erosion and patches on gills. Fungus feeding on fish carcass and leftover food is responsible for this contagious disease.

Cotton wool disease can be controlled by keeping the aquarium clean and preventing stress such as injury or chilling. It can be treated with fungicidal medication, application of gentian violet and salt bath immersion.

Fish tuberculosis – This contagious disease is extremely dangerous and can destroy an entire aquarium. Even humans can contract this disease that spreads through skin cuts and cracks during tank handling.

Symptoms of this disease include white blotches on the exterior, sunken stomach, loss of appetite and frayed fins. It is recommended to immediately separate the sick cichlid and shift aquarium population to a hospital tank treated with antibiotics like Pimafix and Melafix.

Hole-in-the-head disease – This disease also referred to as hexamita is known to affect fresh water fishes including cichlids. Symptoms include weight loss, loss of appetite and depressions on the head. It is predominantly caused by poor diet and poor quality of water. According to studies, lack of calcium, phosphorous and vitamin C and D could be the cause of this cichlid disease.

As most of the cichlid diseases are caused by poor diet and poor quality of water it is recommended to feed your pets in the right manner and keep the aquarium clean and tidy. Research well and read a lot to know about how to keep your cichlids healthy and happy.